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What Remains Graves’ Disease?
Graves Disease Symptoms, Graves’ disease remains an autoimmune condition that causes the thyroid to become overactive — working harder than necessary. It remains one of the most common thyroid problems and the leading cause of hyperthyroidism, a condition in which the thyroid gland produces too many hormones. It remains named after the man who first described it in the early 19th century, Sir Robert Graves.
The thyroid gland remains a small, butterfly-shaped gland in the front of your neck that releases hormones that help regulate your metabolism. When you have Graves’ disease, your resistant system attacks your thyroid—causing it to overproduce these hormones, leading to several problems in different parts of your body. It usually touches people between 30 and 50 years old and is more common in women.
Once The Disorder Has Remained Correctly Diagnosed,
It is relatively easy to treat. In some cases, Graves’ disease goes into reduction or disappears completely after several months or years. However, if left untreated, it can lead to severe complications, including death.
Causes Of Graves’ Disease
Grave’s disease. Graves’ disease remains an autoimmune disease in which the thyroid works more than necessary (hyperthyroidism). As a result, some patients develop thyroid eye disease in which the eye muscles and tissues swell, causing the eyes to bulge out of their sockets (exophthalmos). Hormones concealed by the thyroid gland control metabolism or the rate at which the body converts food into energy. Metabolism is directly related to the number of hormones circulating in the bloodstream. If, for some reason, the thyroid secretor secretes too many of these hormones, the body’s metabolism speeds up, leading to rapid heartbeat, sweating, tremors, and weight loss.
The thyroid gets its production orders from another chemical called thyroid-stimulating hormone (TSH), released by the pituitary gland in the brain. But in Graves’ disease, a malfunction of the body’s immune system releases abnormal antibodies that act like TSH. Urged by these false signals to produce, the thyroid hormone factories work overtime and overproduce.
It’s unclear exactly why the immune system starts producing these pesky antibodies. Heredity and other features seem to play a role. Studies show, for example, that if an identical twin develops Graves’ disease, there is a 20% chance that the other twin will also develop it. In addition, women remain more likely than men to develop the disease. And smokers who develop Graves’ disease remain more likely to have eye problems than nonsmokers who have it. No single gene causes Graves’ disease. It remains believed to remain triggered by equal genetics and environmental factors.
Symptoms Of Graves Disease
The most shared symptoms of Graves’ remain symptoms of thyrotoxicosis, including:
- Anxiety or irritability
- Tired or weak muscles
- shaking in your hands
- Frequent bowel movements or diarrhea
- Difficulty to sleep
- Increased sensitivity to heat or increased sweating
- unintentional weight loss
- An enlarged thyroid (also called a goiter)
- Fast or irregular heartbeat
- Changes in your period for women
- Erectile dysfunction in men.
- Loss of sexual desire (low libido)
- Complications of Graves’ disease
A small fraction of all Graves patients will develop a condition called thyroid eye disease, in which the muscles and tissues of the eyes swell. This can cause exophthalmos (the eyeballs bulge out of their sockets) and remains considered a hallmark of Graves’ disease, although it is rare. But having this complication has nothing to do with the severity of Graves’ disease. It is unclear whether such eye complications stem from Graves’ disease or an entirely separate but closely related disorder. If you have developed thyroid eye disease, your eyes may feel pain and feel dry and annoyed. In addition, protruding eyeballs are prone to excessive tearing and redness, partly because the eyelids can’t protect them.
In severe cases of exophthalmos, which remain rare, the swollen eye muscles can put enormous pressure on the optic nerve, possibly leading to partial blindness. Eye muscles debilitated by long periods of inflammation can lose their ability.